Boldin
Alexander Boldin, Kempten DE
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20110009204 | Golf swing training device - The golf swing training device interactively and passively guides a golf club throughout a training swing by a user from a golfer's stance up to an end of a backswing, and through a forward swing up to a ball impact zone, and that allows a user to hit a ball without any hindrance from the guiding components by naturally swinging follow-though. The guiding components are a guiding-arm, a short lead thereof connected to the distal end of a golf club tows interactively the club shaft up to the end of the backswing, and a resistance-arm, a finger thereof resists passively to the golf club movements throughout the backswing. A speed of rotation of the guiding-arm controlled by a computer system defines a pace of golf club movements, correspondingly a rhythm of the backswing, while a speed of rotation of said resistance-arm defines positioning of the club shaft and, correspondingly, the user's wrists positions relative to the user's forearms at each interactive moment of the backswing. Both the arms are rotatable around an axis set perpendicularly to a swing plane of the club head, at a user's hips height in a housing in which a driving unit is mounted to provide rotation of both the arms. During a forward swing the resistance-arm is working as an interactive guiding component for interactive guiding the golf club when a resistance-arm finger pushing interactively against a golf club shaft at a point near to a club grip and the guiding-arm, in its turn, resisting passively to the golf club movements by the lead, which will be released from the guiding-arm by means of an trigger mechanism at a defined point of the swing controlled by the computer system while the resistance-arm finger continues to push on the club shaft through a hitting portion of the swing, thereby enabling the user to accelerate the swing motion and hit a ball without any hindrance from the guiding components by naturally swinging follow-though. | 01-13-2011 |
Charles Donald Boldin, Oxford, MI US
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20100263179 | LINING FOR BLOCKING WI-FI, ULTRA-SOUND, LASER, VHF, UHF, BLUE TOOTH, AND RFlD TAG SIGNAL - A method of blocking the signal of an RFID tag from being read without consent or knowledge of the owner. A lining or cloth is first made of non-magnetic material containing non-magnetic metal in such concentration as is capable of blocking or attenuating transmission of a signal to or from said Radio Frequency Identification Tag. The lining or cloth is then incorporated into an article of manufacture wherein the article may be used to hold a Radio Frequency identification Tag. | 10-21-2010 |
Ivan Boldin, Moscow RU
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20120193529 | ION CYCLOTRON RESONANCE MEASURING CELLS WITH HARMONIC TRAPPING POTENTIAL - The invention relates to devices and methods for the acquisition of mass spectra with very high mass resolution in ion cyclotron resonance mass spectrometers and methods to produce the devices. The invention presents cylindrical ICR measuring cells with special electrode geometries to generate harmonic trapping potentials for orbiting ions up to the walls of the cell. Only a single DC trapping voltage has to be applied to create the harmonic trapping potential distribution. The sheath of the cylindrical cell is divided by longitudinal gaps into a multitude of sheath electrodes, which either have to carry layers with resistance profiles able to generate parabolic voltage profiles along the sheath electrodes, or which form sheath electrodes of varying width by parabolic gaps, able to create complicated potential distributions which are harmonic on average for orbiting ions. Orbiting ions of a given mass m/z can oscillate harmonically in axial direction with exactly the same oscillation frequency, independent of the radius of their orbit and of their axial oscillation amplitude. Ideally, the cylinders are closed by endcaps with rotationally hyperbolic form, divided into partial electrodes like in infinity cells. The ions can then be excited to their cyclotron motions by dipolar excitation fields also uniformly filling the ICR cell up to the endcaps. The ion clouds orbiting on their cyclotron trajectory are kept together for much longer periods than was possible hitherto, even if they orbit near the sheath electrodes. The image currents thus give rise to minute-long transients, from which mass spectra with ultrahigh mass resolution can be obtained. | 08-02-2012 |
Mark Boldin, Rehovot IL
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20090291888 | MODULATORS OF TNF RECEPTOR ASSOCIATED FACTOR (TRAF), THEIR PREPARATION AND USE - A DNA sequence encoding a protein capable of binding to a tumor necrosis factor receptor-associated factor (TRAF) molecule, TRAF-binding proteins, their isoforms, analogs, fragments and derivatives encoded by the DNA sequence, their methods for the production of the DNA sequences and proteins, and the uses for the DNA sequence and proteins. | 11-26-2009 |
Mark Boldin, Pasadena, CA US
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20080300211 | MICRORNA INHIBITION FOR THE TREATMENT OF INFLAMMATION AND MYELOPROLIFERATIVE DISORDERS - The present disclosure relates to the finding that microRNA-155 plays a role in inflammation, hematopoiesis and myeloproliferation, and that dysregulation of microRNA-155 expression is associated with particular myeloproliferative disorders. Disclosed herein are methods and compositions for diagnosing and treating disorders, including inflammation and myeloproliferation, modulating the levels of expression of one or more genes selected from the group consisting of Cutl1, Arnt1, Picalm, Jarid2, PU.1, Csf1r, HIF1α, Sla, Cepbβ, and Bach1, and the like. | 12-04-2008 |
20090203136 | MODULATING IMMUNE SYSTEM DEVELOPMENT AND FUNCTION THROUGH MICRORNA MIR-146 - The present disclosure relates to the finding that microRNA-146 plays a role in modulating the development and function of the immune system. Immune cell development and function can be modulated by delivery of microRNA-146 (miR-146) or antisense miR-146 to target immune cells or precursor cells. For example, in some embodiments, activity and/or proliferation of certain immune cells is regulated by administering miR-146 oligonucleotides or anti-miR-146 oligonucleotides. In other embodiments, pro-inflammatory cytokine expression in immune cells is regulated by administering a miR-146 oligonucleotide or anti-miR-146. In further embodiments, methods of regulating macrophage activity using antisense miR-146 are provided. Additional methods and compositions for regulating immune system function and development using miR-146 are disclosed. | 08-13-2009 |
20110258716 | MODULATING IMMUNE SYSTEM DEVELOPMENT AND FUNCTION THROUGH MICRORNA MIR-146 - The present disclosure relates to the finding that microRNA-146 plays a role in modulating the development and function of the immune system. Immune cell development and function can be modulated by delivery of microRNA-146 (miR-146) or antisense miR-146 to target immune cells or precursor cells. For example, in some embodiments, activity and/or proliferation of certain immune cells is regulated by administering miR-146 oligonucleotides or anti-miR-146 oligonucleotides. In other embodiments, pro-inflammatory cytokine expression in immune cells is regulated by administering a miR-146 oligonucleotide or anti-miR-146. In further embodiments, methods of regulating macrophage activity using antisense miR-146 are provided. Additional methods and compositions for regulating immune system function and development using miR-146 are disclosed. | 10-20-2011 |
20140220049 | MICRORNA INHIBITION FOR THE TREATMENT OF INFLAMMATION AND MYELOPROLIFERATIVE DISORDERS - The present disclosure relates to the finding that microRNA-155 plays a role in inflammation, hematopoiesis and myeloproliferation, and that dysregulation of microRNA-155 expression is associated with particular myeloproliferative disorders. Disclosed herein are methods and compositions for diagnosing an treating disorders, including inflammation and myeloproliferation, modulating the levels of expression of one or more genes selected from the group consisting of Cutl1, Arntl, Picalm, Jarid2, PU.1, Csflr, HIFlα, Sla, Cepbβ, and Bach1, and the like. | 08-07-2014 |